What causes monomorphic VT?
What causes monomorphic VT?
Monomorphic VT is most commonly seen in patients with underlying structural heart disease. There is typically a zone of slow conduction, most commonly the result of scarring or fibrillar disarray. Causes include prior infarct, any primary cardiomyopathy, surgical scar, hypertrophy, and muscle degeneration.
What causes catecholaminergic polymorphic ventricular tachycardia?
CPVT is caused by a gene defect (abnormal gene). Genes are part of your DNA, the material passed down from parents to children. It can be passed down through families and inherited in different ways (from either one or both parents). But the affected child could be the first case in the family. .
What is the most common cause of ventricular tachycardia?
Sometimes it is not known what causes ventricular tachycardia, especially when it occurs in young people. But in most cases ventricular tachycardia is caused by heart disease, such as a previous heart attack, a congenital heart defect, hypertrophic cardiomyopathy, dilated cardiomyopathy, or myocarditis.
What’s the difference between monomorphic and polymorphic ventricular tachycardia?
VT is considered sustained if it is continuous for at least 30 seconds. If the QRS complex has primarily a single morphology, the VT is monomorphic, whereas if the QRS complex varies, the VT is polymorphic.
What are monomorphic PVCs?
• A high burden of PVCs (>10 percent of all heartbeats, which. increases the risk for congestive heart failure) • Monomorphic PVCs (meaning all of the same morphology. on ECG)
What is monomorphic VT?
INTRODUCTION. Sustained monomorphic ventricular tachycardia (SMVT) is defined by the following characteristics: ●A regular wide QRS complex (≥120 milliseconds) tachycardia at a rate greater than 100 beats per minute. ●The consecutive beats have a uniform and stable QRS morphology.
What the difference between monomorphic and polymorphic ventricular tachycardia?
What is monomorphic ventricular tachycardia?
What causes VT?
What causes VT? VT is produced in two ways: An abnormal heart circuit within the ventricular muscle that is set in motion or triggered (called reentry) Abnormal ventricular muscle that becomes self activated (automaticity)
What does monomorphic VT look like?
Very broad QRS complexes (~ 200 ms) with uniform morphology. Fusion and capture beats are seen in the rhythm strip. Brugada’s sign is present: the time from the onset of the QRS complex to nadir of S wave is > 100 ms (best seen in V6)